The Segal Lab has published a new (July 16, 2018) paper in the Journal of Neuroinflammation detailing evidence of a previously undiscovered mechanism in which cytokines IFNγ and CXCL2 interact to regulate central nervous system lesion location in an experimental allergic encephalomyelitis (EAE) mouse model. You can read the full abstract via PubMed at this link.
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Most Recent Manuscripts
- Enhancing natural killer cells is beneficial in multiple sclerosis - Commentary.
- CNS-resident classical DCs play a critical role in CNS autoimmune disease.
- An IFNγ/CXCL2 regulatory pathway determines lesion localization during EAE.
- A randomized, subject and rater-blinded, placebo-controlled trial of dimethyl fumarate for obstructive sleep apnea.
- Modulation of the Innate Immune System: A Future Approach to the Treatment of Neurological Disease.