The Segal Lab has published a new (July 16, 2018) paper in the Journal of Neuroinflammation detailing evidence of a previously undiscovered mechanism in which cytokines IFNγ and CXCL2 interact to regulate central nervous system lesion location in an experimental allergic encephalomyelitis (EAE) mouse model. You can read the full abstract via PubMed at this link.